Brainz Power CEO, Andrew Kozlovski, Featured In The Huffington Post

The following is a link to Brainz Power's second feature in the Huffington Post in the past month. Learn more about our CEO and his journey of growing Brainz Power in an effort to help all college students suffering from the Adderall epidemic. Link below.

Message From Our  CEO: "I am extremely honored to be featured in the Huffington Post for my achievements in entrepreneurship. Huge thank you to Arianna Huffington and The Post for such an amazing article. I started the company with a mission to address a very real problem that I witnessed first hand as a college student, and its an honor to have such a prestigious media outlet acknowledge my journey. However, we can always get better, and having press like this helps spread the message that students have an alternative and don't have to depend on dangerous and addictive drugs to perform better in school. Thank you all for your support, and thank you to all my loyal customer none of this is possible without you." -Andrew Kozlovski

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Brainz Power Featured in The Huffington Post

Brainz Power along with out CEO's story of founding the company have been featured in the prestigious news source the Huffington Post. Read the full article below. 

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How To Become a Brainz Power Affiliate

1. Go to the following link:

2. Make an account, and start making 20% commissions from each bottle of Brainz Power you sell!

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New Article About Brainz Power's Ingredient BACOPA MONNIERI LEAF & HERBAL EXTRACT

Specifically, in the last 50 years, Bacopa monnieri has been the focus of a great deal of scientific research on brain health, memory improvement, cognitive enhancement, and anxiety reduction.  This recent science is pointing to bacopa monnieri as one of the most powerful and natural nootropic substances you can find. We would speculate this is why you see it used in such a wide variety of traditional treatments.

The first is that bacopa is considered an adaptogen. You can think of adaptogen as any ingredient or chemical which helps your body overcome the effects of stress.

The second pathway is the ability of the bacosides (the active chemical compounds found in bacopa) to help improve the ability of your brain’s neurons to communicate. Bacosides also help promote better kinase functioning in your brain (the enzyme process of breaking down substances to be used by your brain cells). Think of protein kinases as the hallway monitor for your brain. They are the key regulators of cellular functioning.

With improved protein kinases you get help in repairing damaged brain cells and a boost in the creation of new brain cells which ultimately pays off with increased nerve impulse transmission (all hands on deck and working hard).

Bacopa’s primary role in helping improve your memory is by enhancing the way your brain communicates. Primarily, the bacosides delivered from bacopa help with the growth and repair of your brain’s nerve endings called dendrites. With more and better performing dendrites your brain’s ability to retain and recall memories should improve.

Bacopa also has a great effect on reducing anxiety and the incredibly harmful effects of chronic stress on your brain. We believe this is one of the most dangerous symptoms facing the world’s population today.

Conclusion:”There is some evidence to suggest that Bacopa improves memory free recall with evidence for enhancement in other cognitive abilities currently lacking perhaps due to inconsistent measures employed by studies across these cognitive domains. Research into the nootropic effects of Bacopa is in its infancy, with research still yet to investigate the effects of Bacopa across all human cognitive abilities. ”

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Science and Benefits Behind 1 of Brainz Power's 9 ingredients- N-Acetyl-L Carnitine HCI

The following Study is done by the US National Library of Medicine,

Mitochondria produce reactive oxygen species that may contribute to vascular dysfunction. Alpha-lipoic acid and acetyl-L-carnitine reduce oxidative stress and improve mitochondrial function. In a double blind, crossover study, we examined the effects of combined alpha-lipoic acid /acetyl-L-carnitine treatment and placebo (eight weeks per treatment) on vasodilator function and blood pressure in 36 subjects with coronary artery disease. Active treatment increased brachial artery diameter by 2.3% (P=0.008), consistent with reduced arterial tone. Active treatment tended to decrease systolic blood pressure for the whole group (P=0.07) and had a significantly effect in the subgroup with blood pressure above the median (151±20 to 142±18 mmHg, P=0.03) and in the subgroup with the metabolic syndrome (139±21 to 130±18 mmHg, P=0.03). Thus, mitochondrial dysfunction may contribute to the regulation of blood pressure and vascular tone. Further studies are needed to confirm these findings and determine the clinical utility of alpha-lipoic acid/acetyl-L-carnitine as antihypertensive therapy.

In addition to serving as the site of oxidative phosphorylation, it is now clear that mitochondria regulate many cellular functions, in part, by producing reactive oxygen species that signal the adaptive response to environmental stress and injury. Excess production of mitochondria-derived oxidants, however, may have maladaptive effects., There is growing evidence that disturbances of mitochondrial function, including increased oxidant production and altered mitochondria-dependent signaling contribute to the pathogenesis of vascular disease in atherosclerosis and cardiovascular risk factors.,,

On the basis of these observations, we hypothesized that an intervention designed to improve mitochondrial function would have beneficial vascular effects in patients with cardiovascular disease. Recent experimental studies have shown that administration of alpha-lipoic acid and/or acetyl-L-carnitine can reduce oxidant production and improve mitochondrial function in models of aging, Furthermore, these compounds reduce blood pressure and improve endothelial function in animal models of hypertension- and diabetes- The present study was designed to examine the effects of combined alpha-lipoic acid/acetyl-L-carnitine treatment on vascular function and blood pressure in patients with coronary artery disease.

This randomized, placebo-controlled, double blind crossover study demonstrated that combined alpha-lipoic acid/acetyl-L-carnitine treatment was associated with an increase in baseline brachial artery diameter. Furthermore, we observed a non significant trend for a blood pressure lowering effect of alpha lipoic acid/acetyl-L-carnitine in all subjects and a significant reduction in systolic blood pressure in subjects with systolic blood pressures above the median and in subjects with the metabolic syndrome. These findings suggest the possibility that these mitochondria-directed antioxidants reduce basal arterial tone, particularly in two clinically relevant subgroups. In contrast, we observed no effect of treatment on the dilator responses to increased flow, nitroglycerin, or ischemia (reactive hyperemia).

A prior study demonstrated a decrease in systolic blood pressure and a direct vasodilator effect in nailfold capillaries after treatment with oral L-carnitine (3g/d for 20days) in patients with digital vasospastic disease.An open-label study of patients with diabetic nephropathy, reported that long-term alpha lipoic acid treatment (600 mg/d for 18 months) prevented the increases in blood pressure and urine albumin concentration observed in control patients. Experimental studies have consistently demonstrated an anti-hypertensive effect of alpha-lipoic acid or L-carnitine in various rat models of hypertension, including spontaneously hypertensive rats,, uninephrectomized deoxycorticosterone acetate-salt (DOCA-salt) hypertensive ratsand salt-loaded Dahl and Wistar-Kyoto rats.,

Our study differs from several previous human studies that examined the effects of lipoic acid or carnitine on endothelial function. For example, Heitzer and colleagues observed an acute improvement in endothelium-dependent dilation of forearm microvessels following an intra-arterial infusion of lipoic acid (final concentration 0.2 mmoles/L) in patients with diabetes mellitus. Sola and colleagues recently reported improved brachial artery flow-mediated dilation following treatment with lipoic acid 300 mg/d for four weeks in young patients with the metabolic syndrome (mean age 46 years). Intravenous administration of L-carnitine (3 gram bolus) enhanced reactive hyperemia in patients with peripheral arterial disease. The apparent discrepancies between the results of those prior studies and the present study likely relate to the marked differences in dose, route of administration, vascular bed studied and/or patient population.

The mechanisms accounting for the increased brachial artery diameter and suggestive anti-hypertensive effects of alpha-lipoic acid and acetyl-L-carnitine in our study remain undefined. We observed no effect of treatment on serum lipids, glucose, and insulin, which might have influenced endothelial function or arterial diameter. Experimental studies indicate that alpha-lipoic acid and acetyl-L-carnitine play important and potentially synergistic roles in normal mitochondrial function, and that reduced levels of these compounds are associated with increased mitochondrial oxidant production.,, In addition, lipoic acid supplementation has favorable effects on cellular redox state and has been shown to decrease lipid peroxidation and cellular production of reactive oxygen species.,,, The effects of alpha-lipoic acid and acetyl-L-carnitine on oxidative stress which contributes to the pathogenesis and cardiovascular complications of hypertension suggests that these compounds may be useful adjuncts in treatment. In the present study, we investigated the possibility that these compounds reduced oxidative stress and inflammation, but observed no effect of treatment on urinary F2 isoprostanes or serum C-reactive protein. It is important to point out, however, that these systemic markers may not accurately reflect events in the vascular wall.

Despite the effects on blood pressure and basal diameter, it is notable that we only observed a trend for increased total serum carnitine following treatment. It is known that acetyl-L-carnitine and alpha-lipoic acid are rapidly metabolized in human subjects with plasma half lives of 4.2 hours and 30 minutes,respectively. Thus, it is likely that acetyl-L-carnitine and lipoic acid metabolites and/or tissue levels may be more relevant for the observed effects. Similarly, the lack of effect of treatment on urine F2 isoprostanes does not rule out an effect of active treatment on oxidative stress at the tissue level.

We observed a particular benefit of alpha-lipoic acid/acetyl-L-carnitine in patients with the metabolic syndrome. This syndrome of insulin resistance is associated with hypertension; improvement of insulin sensitivity could have an anti-hypertensive effect. Consistent with our findings, several experimental studies suggest a blood pressure-lowering effect in models of diabetes mellitus or insulin resistance.- Those results and our findings are consistent with the growing evidence linking insulin resistance to mitochondrial dysfunction

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Andrew Kozlovski Brainz Power Huffington Post
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